Which of the following is NOT a cause of intrahepatic jaundice?

Intrahepatic jaundice occurs when there is an impairment of bilirubin metabolism within the liver itself, which leads to an accumulation of bilirubin in the blood. Conditions that cause intrahepatic jaundice typically involve liver cell dysfunction or damage.

Dubin-Johnson syndrome and Crigler-Najjar syndrome are both genetic disorders that affect the liver's ability to process bilirubin. Dubin-Johnson syndrome is due to a defect in bilirubin transport, leading to the conjugated form of bilirubin accumulating in the liver and subsequently leaking into the bloodstream. Crigler-Najjar syndrome, on the other hand, is characterized by a deficiency in the enzyme that conjugates bilirubin, leading to an accumulation of unconjugated bilirubin in the bloodstream. Both of these syndromes are clear examples of conditions that can cause intrahepatic jaundice.

Hepatitis is an inflammation of the liver, often caused by viral infections, alcohol, or toxins. This inflammation can disrupt the normal processing of bilirubin, leading to an increase in bilirubin levels in the bloodstream and resulting in intrahepatic jaundice.

In contrast, choledocholithiasis refers to the presence of gallstones in the common bile duct, leading to blockage